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PAUL MASON | HIGH BLOOD PRESSURE? Not salt! LACK of nitric oxide…due to fructose!
presents episode 982 | Dr Paul Mason
BNS podcast

In 2017, the Australian dietary guidelines removed in the upper limits to sodium intake

The fluid volume of your blood is covered by sodium levels.
If not enough: feel thirsty, still drink a lot, but without sodium, blood will not hold fluids, so will urinate them out

The fluid volume of your blood is governed by sodium levels:
people on a ketogenic diet often need more salt, so if constantly thirsty, and need to urinate: it is likely of sodium deficiency

When sodium deficient, may feel dizzy, when quickly standing: simply add more sodium to your diet

Fructose directly contributes to insulin resistance: sugar, which is 50% fructose, and 50% glucose,

In a study of children, two groups, one: typical sugar carbohydrates to: replaced fructose with 100% glucose
Results: glucose group, within a week, significantly reduced fat in the liver

Fatty liver is a direct cause of insulin resistance: insulin not working properly, so body will increase insulin

Insulin resistance is very tissue specific: while insulin may stop working well in muscle tissue,
it could still work well in the fat tissue: storing sugar is fat and work well in the kidneys: hold onto sodium!

This is a much more likely factor of colon insulin not working, well

Kidneys hold on to sodium: high blood pressure.

On the other hand, you can consume a lot of sodium, if you have low sodium, then you will simply urinate excess sodium out

Fructose causes high blood pressure: primarily through insulin resistance
It can also interact with nitric oxide through uric acid, Dr. Robert Lustig’s work

Dr. Robert Lustig:
Fructose metabolism, leads to changes in uric acid levels, which lead to interruptions in uric acid

Nitric oxide relaxes blood vessels, so when interrupted, leads to high blood pressure and ED

“I believe predominate cause of blood pressure rise is: sodium, retaining effects of insulin at the kidneys”


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